Fellowships: Neurodevelopment, Oxidative Stress and Synaptic Plasticity – F03A
*New
The F03A study section reviews fellowship applications on the basic cellular and molecular biology of neuronal, glial, retinal and other excitable cells. Studies proposed in these applications may examine the fundamental mechanisms of neuronal cell function, including mechanisms relevant to specific disease processes.
Applications examining the molecular and cellular processes leading to cell death as well as cell specification, differentiation and development including mechanisms that specify or influence migratory events are also reviewed here. Studies examining aging, degeneration and regeneration of neuronal connectivity as well as the consequences of exposure to psychoactive drugs on the above processes are also reviewed in F03A.
The List of Reviewers lists all present, whether standing members or temporary, to provide the full scope of expertise present on that date. Lists are posted 30 days before the meeting and are tentative, pending any last minute changes.
Review Dates
Topics
- The cellular and molecular mechanisms of synaptogenesis and synaptic plasticity
- The cellular and molecular mechanisms by which extra- and intracellular signals are transduced in neural and excitable cells
- Neuronal trafficking including membrane vesicle trafficking, cytoskeleton and cell adhesion molecules
- Neural development and molecular mechanisms impacting the related diseases/disorders
- Proliferation, differentiation, and migration of neural/glial progenitors and stem cells, including cell lineage, cell fate specification, and neural patterning
- Cellular and molecular mechanisms of axon and dendritic guidance, outgrowth and regeneration
- Molecular mechanisms underlying retinal development and degeneration
- Genetic and epigenetic mechanisms, and the regulation of gene expression
- Myelin and glial biology, including neuroinflammation, myelination and remyelination
- Redox biology in the nervous system, molecular mechanisms of the production of reactive oxygen species (ROS) and oxidative stress
